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The transsulfuration pathway is irreversible from cysteine and is reported to only occur in instances when levels of methionine are biologically dispensable but is also regulated by the availability of CSE and a biological need for cysteine (Homocysteine metabolism is reviewed in depth by Azzini et al, 2020)

Via the STAT3/GPX4 pathway, Thiostrepton (TST) induces ferroptosis in pancreatic cancer cells by reducing cellular iron overload, increasing ROS accumulation, raising MDA levels, and decreasing GSH-PX in these cells [138]